参考文献/References:
[1] THI M T T,WIBOWO D,REHM B H A.Pseudomonas aeruginosa biofilms[J].Int J Mol Sci,2020,21(22):8671.
[2] ZILBERBERG M D,NATHANSON B H,SULHAM K,et al.A novel algorithm to analyze epidemiology and outcomes of carbapenem resistance among patients with hospital-acquired and ventilator-associated pneumonia:a retrospective cohort study[J].Chest,2019,155(6):1119-1130.
[3] KOCSIS B,GULYS D,SZAB D.Diversity and distribution of resistance markers in Pseudomonas aeruginosa international high-risk clones[J].Microorganisms,2021,9(2):359.
[4] JURADO-MARTN I,SAINZ-MEJAS M,MCCLEAN S.Pseudomonas aeruginosa:an audacious pathogen with an adaptable arsenal of virulence factors[J].Int J Mol Sci,2021,22(6):3128.
[5] YAHR T L,BARBIERI J T,FRANK D W.Genetic relationship between the 53- and 49-kilodalton forms of exoenzyme S from Pseudomonas aeruginosa[J].J Bacteriol,1996,178(5):1412-1419.
[6] HORNA G,RUIZ J.Type 3 secretion system of Pseudomonas aeruginosa[J].Microbiol Res,2021,246:126719.
[7] 张鸿,陈炜.铜绿假单胞菌Ⅲ型分泌系统结构及调控机制的研究进展[J].中国感染与化疗杂志,2020,20(3):335-339.
ZHANG H,CHEN W.Research updates on the structure and regulatory mechanism of type Ⅲ secretion system in Pseudomonas aeruginosa[J].Chin J Infect Chemother,2020,20(3):335-339.
[8] HARDY K S,TUCKEY A N,RENEMA P,et al.ExoU induces lung endothelial cell damage and activates pro-inflammatory caspase-1 during Pseudomonas aeruginosa infection[J].Toxins (Basel),2022,14(2):152.
[9] HARDY K S,TESSMER M H,FRANK D W,et al.Perspectives on the Pseudomonas aeruginosa type Ⅲ secretion system effector ExoU and its subversion of the host innate immune response to infection[J].Toxins (Basel),2021,13(12):880.
[10] FOULKES D M,MCLEAN K,ZHENG Y,et al.A pipeline to evaluate inhibitors of the Pseudomonas aeruginosa exotoxin U[J].Biochem J,2021,478(3):647-668.
[11] TESSMER M H,DECERO S A,DEL ALAMO D,et al.Charac-terization of the ExoU activation mechanism using EPR and integrative modeling[J].Sci Rep,2020,10(1):19700.
[12] RAO L,DE LA ROSA I,XU Y,et al.Pseudomonas aeruginosa survives in epithelia by ExoS-mediated inhibition of autophagy and mTOR[J].EMBO Rep,2021,22(2):e50613.
[13] VOTH S,GWIN M,FRANCIS C M,et al.Virulent Pseudomonas aeruginosa infection converts antimicrobial amyloids into cytotoxic prions[J].FASEB J,2020,34(7):9156-9179.
[14] KHODAYARY R,NIKOKAR I,MOBAYEN M R,et al.High incidence of type Ⅲ secretion system associated virulence factors (exoenzymes) in Pseudomonas aeruginosa isolated from Iranian burn patients[J].BMC Res Notes,2019,12(1):28.
[15] KROKEN A R,GAJENTHRA KUMAR N,YAHR T L,et al.Exotoxin S secreted by internalized Pseudomonas aeruginosa delays lytic host cell death[J].PLoS Pathog,2022,18(2):e1010306.
[16] ARMENTROUT E I,KUNDRACIK E C,RIETSCH A.Cell-type-specific hypertranslocation of effectors by the Pseudomonas aeruginosa type Ⅲ secretion system[J].Mol Microbiol,2021,115(2):305-319.
[17] GARAI P,BERRY L,MOUSSOUNI M,et al.Killing from the inside:intracellular role of T3SS in the fate of Pseudomonas aeruginosa within macrophages revealed by mgtC and oprF mutants[J].PLoS Pathog,2019,15(6):e1007812.
[18] PARK T.Crk and CrkL as therapeutic targets for cancer treatment[J].Cells,2021,10(4):739.
[19] FU Q,YU Z.Phosphoglycerate kinase 1 (PGK1) in cancer:a promising target for diagnosis and therapy[J].Life Sci,2020,256:117863.
[20] MOHAMED M F,GUPTA K,GOLDUFSKY J W,et al.CrkII/Abl phosphorylation cascade is critical for NLRC4 inflammasome activity and is blocked by Pseudomonas aeruginosa ExoT[J].Nat Commun,2022,13(1):1295.
[21] MOHAMED M F,WOOD S J,ROY R,et al.Pseudomonas aeruginosa ExoT induces G1 cell cycle arrest in melanoma cells[J].Cell Microbiol,2021,23(8):e13339.
[22] MANCL J M,SUAREZ C,LIANG W G,et al.Pseudomonas aeruginosa exoenzyme Y directly bundles actin filaments[J].J Biol Chem,2020,295(11):3506-3517.
[23] BELYY A,MERINO F,MECHOLD U,et al.Mechanism of actin-dependent activation of nucleotidyl cyclase toxins from bacterial human pathogens[J].Nat Commun,2021,12(1):6628.
[24] WAGENER B M,ANJUM N,CHRISTIAANS S C,et al.Exoenzyme Y contributes to end-organ dysfunction caused by Pseudomonas aeruginosa pneumonia in critically ill patients:an exploratory study[J].Toxins (Basel),2020,12(6):369.
[25] SILISTRE H,RAOUX-BARBOT D,MANCINELLI F,et al.Prevalence of ExoY activity in Pseudomonas aeruginosa reference panel strains and impact on cytotoxicity in epithelial cells[J].Front Microbiol,2021,12:666097.
[26] 赵轩,江晓烽,秦江雷,等.载铜绿假单胞菌OprF和PcrV基因联合DNA疫苗的水凝胶缓释系统的构建及免疫效力评价[J].解放军医学杂志,2022,47(9):871-878.
ZHAO X,JIANG X F,QIN J L,et al.Construction and immune efficacy evaluation of a hydrogel sustained-release system containing a combined DNA vaccine of Pseudomonas aeruginosa OprF and PcrV genes[J].Med J Chin PLA,2022,47(9):871-878.
[27] TRIGGS C T,TANSKI J M.Crystallographic and spectroscopic characterization of 3-chloro-5-fluoro-salicyl-aldehyde[J].Acta Crystallogr E Crystallogr Commun,2020,76(Pt 12):1810-1812.
[28] SHARMA P,ELOFSSON M,ROY S.Attenuation of Pseudomonas aeruginosa infection by INP0341,a salicylidene acylhydrazide,in a murine model of keratitis[J].Virulence,2020,11(1):795-804.
[29] MOIR D T,BOWLIN N O,BERUBE B J,et al.A structure-function-inhibition analysis of the Pseudomonas aeruginosa type Ⅲ secretion needle protein PscF[J].J Bacteriol,2020,202(18):e00055-e00074.
[30] ZHAO X,YU Z,DING T.Quorum-sensing regulation of antimicrobial resistance in bacteria[J].Microorganisms,2020,8(3):425.
[31] GARCA-REYES S,SOBERN-CHVEZ G,COCOTL-YANEZ M.The third quorum-sensing system of Pseudomonas aeruginosa:pseudomonas quinolone signal and the enigmatic PqsE protein[J].J Med Microbiol,2020,69(1):25-34.
[32] PENA R T,BLASCO L,AMBROA A,et al.Relationship between quorum sensing and secretion systems[J].Front Microbiol,2019,10:1100.
[33] SCHTZ C,HO D K,HAMED M M,et al.A new PqsR inverse agonist potentiates tobramycin efficacy to eradicate Pseudomonas aeruginosa biofilms[J].Adv Sci (Weinh),2021,8(12):e2004369.
[34] WARRIER A,SATYAMOORTHY K,MURALI T S.Quorum-sensing regulation of virulence factors in bacterial biofilm[J].Future Microbiol,2021,16:1003-1021.
[35] ZHANG P,GUO Q,WEI Z,et al.Baicalin represses type three secretion system of Pseudomonas aeruginosa through PQS system[J].Molecules,2021,26(6):1497.
[36] WEI Q,BHASME P,WANG Z,et al.Chinese medicinal herb extract inhibits PQS-mediated quorum sensing system in Pseudomonas aeruginosa[J].J Ethnopharmacol,2020,248:112272.
[37] LEE V T,PUKATZKI S,SATO H,et al.Pseudolipasin A is a specific inhibitor for phospholipase A2 activity of Pseudomonas aeruginosa cytotoxin ExoU[J].Infect Immun,2007,75(3):1089-1098.
[38] KIM D,BAEK J,SONG J,et al.Identification of arylsulfonamides as ExoU inhibitors[J].Bioorg Med Chem Lett,2014,24(16):3823-3825.
[39] MOIR D T,OPPERMAN T J,ARON Z D,et al.Adjunctive therapy for multidrug-resistant bacterial infections:type Ⅲ secretion system and efflux inhibitors[J].Drug Discov Today,2021,26(9):2173-2181.
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