参考文献/References:
[1] LU Z F,LI B,QIAO P,et al.Surgical treatment of traumatic bifrontal contusions:when and how[J].World Neurosurg,2016,93(3):261-269.
[2] ABRAHAMSON E E,IKONOMOVIC M D.Brain injury-induced dysfunction of the blood brain barrier as a risk for dementia[J].Exp Neurol,2020,328(2):113257-113263.
[3] ZHU B,EOM J,HUNT R F.Transplanted interneurons improve memory precision after traumatic brain injury[J].Nat Commun,2019,10(1):5156-5162.
[4] RAMOS-CEJUDO J,WISNIEWSKI T,MARMAR C,et al.Traumatic brain injury and Alzheimer′s disease:the cerebrovascular link[J].EBio Med,2018,28(5):21-30.
[5] MENDEZ M F.What is the relationship of traumatic brain injury to dementia [J].J Alzheimers Dis,2017,57(3):667-681.
[6] 陆兆丰,贾锋,邱永明,等.亚低温对创伤性脑损伤后线粒体α-酮戊二酸脱氢酶活性的影响[J].中华神经外科杂志,2006,22(11):659-662.
[7] 夏天光,孔宪斌,王明丽,等.光遗传技术调控新生神经元电活动对颅脑损伤小鼠认知功能的影响[J].新乡医学院学报,2018,35(10):858-864.
[8] LAZZARIO G,AMORINI A M,SIGNORETTI S,et al.Pyruvate dehydrogenase and tricarboxylic acid cycle enzymes are sensitive targets of traumatic brain injury induced metabolic derangement[J].Int J Mol Sci,2019,20(22):E5774-E5786.
[9] SHI Q,XU H,YU H,et al.Inactivation and reactivation of the mitochondrial α-ketoglutarate dehydrogenase complex [J].J Biol Chem,2011,286(20):17640-17648.
[10] CHEN H,DENTON T T,XU H,et al.Reductions in the mitochondrial enzyme αketoglutarate dehydrogenase complex in neurodegenerative disease-beneficial or detrimental[J].J Neurochem,2016,139(5):823-838.
[11] BANERJEE K,MUNSHI S,XU H,et al.Mild mitochondrial metabolic deficits by α-ketoglutarate dehydrogenase inhibition cause prominent changes in intracellular autophagic signaling:Potential role in the pathobiology of Alzheimer′s disease[J].Neurochem Int,2016,96(2):32-45.
[12] LOONEN A J,WILFFERT B,IVANOVA S A.Putative role of pharmacogenetics to elucidate the mechanism of tardive dyskinesia in schizophrenia[J].Pharmacogenomics,2019,20(17):1199-1223.
[13] LAI J C,WHITE B K,BUERSTATTE C R,et al.Chronic hypoxia in development selectively alters the activities of key enzymes of glucose oxidative metabolism in brain regions[J].Neurochem Res,2003,28(6):933-940.
[14] PANDYA J D,LEUNG L Y,YANG X,et al.Comprehensive profile of acute mitochondrial dysfunction in a preclinical model of severe penetrating TBI[J].Front Neurol,2019,32(10);605-615.
[15] HUANG H M,OU H C,CHEN H L,et al.Protective effect of α-keto-β-methyl-n-valeric acid on BV-2 microglia under hypoxia or oxidative stress[J].Ann NY Acad Sci,2005,1042(3):272-278.
[16] CHEN Z,ZHONG C.Decoding Alzheimer′s disease from perturbed cerebral glucose metabolism:implications for diagnostic and therapeutic strategies[J].Prog Neurobiol,2013,108(5):21-43.
[17] BUBBER P,HARTOUNIAN V,GIBSON G E,et al.Abnormalities in the tricarboxylic acid (TCA) cycle in the brains of schizophrenia patients[J].Eur Neuropsychopharmacol,2011,21(3):254-260.
相似文献/References:
[1]陈翀,李晓红,涂悦,等.糖原合成酶激酶3对大鼠创伤性脑损伤后认知功能的影响[J].新乡医学院学报,,():000.
[2]陈翀,李晓红,涂悦,等.糖原合成酶激酶3对大鼠创伤性脑损伤后认知功能的影响[J].新乡医学院学报,,():000.
[3]陆兆丰,朱刚毅,郏建臣,等.创伤性脑损伤大鼠半暗带脑组织中Elk1的表达及作用[J].新乡医学院学报,2020,37(2):107.[doi:10.7683/xxyxyxb.2020.02.002]
LU Zhaofeng,ZHU Gangyi,JIA Jianchen,et al.Expression and role of Elk1 in the contusion penumbra tissues of rats with traumatic brain injury[J].Journal of Xinxiang Medical University,2020,37(4):107.[doi:10.7683/xxyxyxb.2020.02.002]
[4]张召宇,李梦欣,李慧堂,等.神经干细胞联合神经营养因子-3基因修饰的嗅鞘细胞移植对创伤性脑损伤大鼠神经功能的影响及机制[J].新乡医学院学报,2023,40(6):501.[doi:10.7683/xxyxyxb.2023.06.001]
ZHANG Zhaoyu,LI Mengxin,LI Huitang,et al.Effect and mechanism of combined transplantation of neural stem cells and olfactory ensheathing cells modified by neurotrophin-3 gene on neural function of rats with traumatic brain injury[J].Journal of Xinxiang Medical University,2023,40(4):501.[doi:10.7683/xxyxyxb.2023.06.001]